1,503 research outputs found

    Challenges for MapReduce in Big Data

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    In the Big Data community, MapReduce has been seen as one of the key enabling approaches for meeting continuously increasing demands on computing resources imposed by massive data sets. The reason for this is the high scalability of the MapReduce paradigm which allows for massively parallel and distributed execution over a large number of computing nodes. This paper identifies MapReduce issues and challenges in handling Big Data with the objective of providing an overview of the field, facilitating better planning and management of Big Data projects, and identifying opportunities for future research in this field. The identified challenges are grouped into four main categories corresponding to Big Data tasks types: data storage (relational databases and NoSQL stores), Big Data analytics (machine learning and interactive analytics), online processing, and security and privacy. Moreover, current efforts aimed at improving and extending MapReduce to address identified challenges are presented. Consequently, by identifying issues and challenges MapReduce faces when handling Big Data, this study encourages future Big Data research

    Nuclear g-Factors of the 1229 and 2911 keV Isomers in 143Nd

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    開始ページ、終了ページ: 冊子体のページ付

    A project on magnetic survey in Bransfield Strait, Antarctic Peninsula

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    第2回極域科学シンポジウム/第31回極域地学シンポジウム 11月16日(水) 国立国語研究所 2階フロ

    Testing real-time systems using TINA

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    The paper presents a technique for model-based black-box conformance testing of real-time systems using the Time Petri Net Analyzer TINA. Such test suites are derived from a prioritized time Petri net composed of two concurrent sub-nets specifying respectively the expected behaviour of the system under test and its environment.We describe how the toolbox TINA has been extended to support automatic generation of time-optimal test suites. The result is optimal in the sense that the set of test cases in the test suite have the shortest possible accumulated time to be executed. Input/output conformance serves as the notion of implementation correctness, essentially timed trace inclusion taking environment assumptions into account. Test cases selection is based either on using manually formulated test purposes or automatically from various coverage criteria specifying structural criteria of the model to be fulfilled by the test suite. We discuss how test purposes and coverage criterion are specified in the linear temporal logic SE-LTL, derive test sequences, and assign verdicts

    Measurement of the CP Violation Parameter sin(2phi_1) in B^0_d Meson Decays

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    We present a measurement of the Standard Model CP violation parameter sin(2phi_1) based on a 10.5 fb^{-1} data sample collected at the Upsilon(4S) resonance with the Belle detector at the KEKB asymmetric e+e- collider. One neutral B meson is reconstructed in the J/psi K_S, psi(2S) K_S, chi_{c1} K_S, eta_c K_S, J/psi K_L or J/psi pi^0 CP-eigenstate decay channel and the flavor of the accompanying B meson is identified from its charged particle decay products. From the asymmetry in the distribution of the time interval between the two B-meson decay points, we determine sin(2phi_1) = 0.58 +0.32-0.34 (stat) +0.09-0.10 (syst).Comment: LaTex, 13 pages, 3 figures, submitted to P.R.

    HuR is exported to the cytoplasm in oral cancer cells in a different manner from that of normal cells

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    HuR, a ubiquitously expressed member of the Hu protein family that binds and stabilizes an AU-rich element (ARE)-containing mRNAs, is known to shuttle between the nucleus and the cytoplasm via several export pathways. When normal cells were treated with heat shock, HuR was exported to the cytoplasm in a chromosome maintenance region 1 (CRM1)-dependent manner. However, in this study, we demonstrate that HuR is exported to the cytoplasm in oral cancer cells even if the cells were treated with the inhibitor of the CRM1-independent export pathway. Immunohistochemical and biochemical analyses showed that HuR existed in both the cytoplasm and the nucleus in oral cancer cells, such as HSC-3 and Ca9.22, but existed entirely inside the nucleus in normal cells. AU-rich element-mRNAs were also exported to the cytoplasm and stabilised in the oral cancer cells, which were inhibited by HuR knockdown. This export of HuR was not affected by at least 7 h of treatment of leptomycin B (LMB), which is an inhibitor of the CRM1-dependent export pathway. These findings suggest that HuR is exported to the cytoplasm in oral carcinoma cells in a different manner from that of normal cells, and is likely to occur through the perturbation of a normal export pathway
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